Short bowel syndrome is a malabsorption disorder whose common causes for extensive bowel resection are volvulus, Crohn's disease, mesenteric infarction, radiculitis, cancer, and congenital abnormalities. Medical treatment of short bowel syndrome is usually parenteral nutrition, oral feeding, and concomitant use of medications.

Short bowel syndrome (SBS) is a malabsorptive condition associated with a high incidence of complications and high utilization of health care resources. Therefore, in some cases, physicians may recommend vitamin and mineral supplementation for patients with this syndrome.

Short bowel syndrome is a malabsorptive condition associated with a high incidence of complications and high utilization of health care resources. Therefore, in some patients, physicians need to control food intake, in which controlling sugar, protein, and fat intake is extremely important.

People with short bowel syndrome need to pay close attention to their diet to improve their symptoms and quickly restore their intestinal health. So, when you have short bowel syndrome, what should you eat?

There are very few published reports on the use of home enteral nutritional support (enteral nutrition) in short bowel syndrome. In a recent report from a large at-home enteral nutrition program in Canada, only 9 of 727 patients received home parenteral nutrition for short bowel syndrome.

Successful treatment of patients with short bowel syndrome (SBS) depends on adaptation of the remaining bowel, which requires a combination of pharmacological and nutritional therapy. Although the short bowel syndrome diet is quite similar for those with and without colonic segments, some key differences should be noted. This article focuses on dietary interventions that promote adaptation, increase absorption, and consequently reduce stool volume.

Dietary therapy is an important component of care for patients with short bowel syndrome. The cornerstone of dietary therapy is the adjustment of food intake to maximize nutrient and fluid utilization by reducing stool output. Fecal output in short bowel syndrome is driven by fluid load exceeding the absorptive capacity of the shortened intestine; but other factors also contribute.